Preliminary experiments show conolidine may possibly inhibit unique ion channels, minimizing neuronal excitability and limiting soreness indicators. This mechanism is particularly applicable in neuropathic discomfort, in which abnormal signaling triggers persistent distress. Moreover, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to suffering perceptio... https://janej279byu2.wannawiki.com/user